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Of course this requires that we ignore my theory, and pretend that AI diseases actually exist.
Am I reading you correctly when I think that you're saying that there is no such thing as AI diseases? Please enlighten me because I am completely baffled Because if you think that there actually is no such thing as an AI disease, than how do you explain a child born with diabetes or who acquires diabetes shortly after birth? Where is the antigen here? What is the trigger and reason other than a defective gene?
Monique[/quote]
Diagnosed 2011 with LC. Currently on Low Dose Naltrexone (LDN)
Since this isn't validated by accepted conventional medical practices, it can only be considered to be a theory at this point, but I truly believe that some day it will be verified as the true cause of, if not all, certainly most syndromes that are currently considered to be autoimmune diseases. For a basic explanation, please see page 225 in my book. It might be beneficial to review the information beginning on about page 220, to provide the background information leading up to that discussion. If you don't have a copy of the book handy, please let me know, and I'll be happy to email you a copy in any format you choose, or if you prefer, I can email a copy of the relevant section of the book that addresses this topic.
Please be aware that issues that exist at birth are almost certainly due to environmental influences (together with genetic and epigenetic contributions) that affected the mother and/or father, but are still likely to be traceable to one or more external antigens (that affected one or both parents (in addition to the child).
Research shows that even GERD, for example, can be shown to meet the criteria of the category of diseases that are classified as having an autoimmune etiology. That strengthens my case, since GERD is certainly treatable (and effectively prevented) by avoiding the foods and/or drugs that are known to trigger it, and by maintaining a sufficient vitamin D level, provided that the treatment is used before the strength of the lower esophageal sphincter (LES) is destroyed by the use of PPIs or other iatrogenic drugs. This is discussed on page 214, with reference to preliminary information that includes the citation of medical research data, back on page 180. Perhaps I should consider writing a book on this topic, because there is a heck of a lot of information available in the medical literature that supports my theory, but everyone continues to ignore the data, because they don't know what to do with it, and they certainly don't want to rock the boat, since it flies in the face of conventional medical treatment of so-called autoimmune diseases.
In most cases, I believe that the original trigger can be traced back to gluten (often in combination with a vitamin D deficiency), except that as I mentioned, in some cases the trace has to go all the way back to parents, and in the case of epigenetic effects, it's a transgenerational effect that goes all the way back to the grandparents (skipping the parents).
And please remember that if an antigen is ignored long enough, at least some of the damage is likely to become permanent, so that eventually, just avoiding the antigen may not be sufficient to completely resolve the problem, (though avoidance should certainly help, proving that the antigen is/was indeed a causative factor). For example, I have a lot of neurological damage that is now permanent, because it took me too long to figure out the problem. And I also have a lot of secondary food sensitivities that are now obviously permanent, because I didn't cut gluten out of my diet many years ago, when doing so could have prevented a lot of subsequent problems.
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
Hi Joe,
I just read your post. Sorry you are dealing with all of this...
I have been thinking of you and hope you and Dr Yost come up with a plan to get you feeling better!
Hopefully you are able to enjoy this weather with Nestle!
Jenny
Because if you think that there actually is no such thing as an AI disease, than how do you explain a child born with diabetes or who acquires diabetes shortly after birth? Where is the antigen here? What is the trigger and reason other than a defective gene?
