Fodmaps (I hope this wasn't posted previously)
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Fodmaps (I hope this wasn't posted previously)
DISCLAIMER: I am not a doctor and don't play one on TV.
LDN July 18, 2014
Joan
LDN July 18, 2014
Joan
It seems that medical professionals will do almost anything, and go to all sorts of extremes to keep from having to admit that non-celiac gluten sensitivity actually exists. 
As if calling it something else and trying to cloud the issue somehow magically changes the fact that they have been wrong for all these years.
Tex
As if calling it something else and trying to cloud the issue somehow magically changes the fact that they have been wrong for all these years.
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
I understand the premise behind this article.Although there are those of us who have non-celiac gluten sensitivity, there are also people who are sensitive to certain carbs and have malabsorption problems who aren't actually reacting to gluten. I don't think you can put it all under one umbrella.
Leah
Leah
Leah,
Just as with the trials associated with oats in the diets of celiacs, I have a hunch that the trials connected with the reintroduction of gluten into their diets did not last long enough. It takes many people a long time to begin to react again after gluten has been removed from their diet for a while.
The experts claim that oats are safe for most celiacs, because the trials they used did not show large numbers of relapses. And yet if you compare this with actual real-life experiences, you find that most celiacs do indeed react to oats. The experts simply do not use trials that last long enough to allow for sufficient development of symptoms in most cases.
Look at the mechanics of the situation. Gluten is the primary cause of leaky gut. Certain bacteria (such as the bacteria that cause cholera) can cause the release of zonulin, but gluten is the only only food element known to promote the release of zonulin (which causes the tight junctions to open). Without leaky gut, you can't have true food sensitivities (you can only have IgE-based reactions. At least that's the way I see it. Obviously I could be wrong, of course.
Tex
Just as with the trials associated with oats in the diets of celiacs, I have a hunch that the trials connected with the reintroduction of gluten into their diets did not last long enough. It takes many people a long time to begin to react again after gluten has been removed from their diet for a while.
The experts claim that oats are safe for most celiacs, because the trials they used did not show large numbers of relapses. And yet if you compare this with actual real-life experiences, you find that most celiacs do indeed react to oats. The experts simply do not use trials that last long enough to allow for sufficient development of symptoms in most cases.
Look at the mechanics of the situation. Gluten is the primary cause of leaky gut. Certain bacteria (such as the bacteria that cause cholera) can cause the release of zonulin, but gluten is the only only food element known to promote the release of zonulin (which causes the tight junctions to open). Without leaky gut, you can't have true food sensitivities (you can only have IgE-based reactions. At least that's the way I see it. Obviously I could be wrong, of course.
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
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Leah,
While what you are suggesting is theoretically possible, the reality is that in order to lose the ability to produce enzymes (assuming one was not born without the ability to produce those enzymes, which would constitute a genetic problem), one has to experience enteritis (inflammation of the small intestine). The ability to produce lactase can be lost relatively quickly (within a matter or a day or so). Even a short bout with the flu virtually always causes a lactase deficiency.
But the ability to produce the other enzymes is lost only if the enteritis continues for a significant length of time, and they are progressively lost (they're not all lost at the same time). In order to lose more than a couple, it appears that one must have severe enteritis for many months (or years), though data on the exact timing is not available, because not much research has been devoted to this issue.
Generally speaking, the only GI issues associated with long-term enteritis are the IBDs. Right? Show me someone who has an IBD who is not sensitive to gluten and I will show you a very rare case, (just look at the statistics in our own test files). The exceptions are usually drug-induced cases, and in those cases, stopping the use of the drug typically stops the inflammation, if it's done before other sensitivities are allowed time to develop.
And examples based on "IBS" won't work, because if long-term enteritis (intestinal inflammation) is involved, then clearly (by definition), you are talking about an inflammatory bowel disease instead. Remember, only serious, long-term intestinal inflammation can cause progressive loss of multiple enzymes.
So it might be possible, but it appears to be very, very unlikely, statistically. At least that's the way I see it.
Tex
While what you are suggesting is theoretically possible, the reality is that in order to lose the ability to produce enzymes (assuming one was not born without the ability to produce those enzymes, which would constitute a genetic problem), one has to experience enteritis (inflammation of the small intestine). The ability to produce lactase can be lost relatively quickly (within a matter or a day or so). Even a short bout with the flu virtually always causes a lactase deficiency.
But the ability to produce the other enzymes is lost only if the enteritis continues for a significant length of time, and they are progressively lost (they're not all lost at the same time). In order to lose more than a couple, it appears that one must have severe enteritis for many months (or years), though data on the exact timing is not available, because not much research has been devoted to this issue.
Generally speaking, the only GI issues associated with long-term enteritis are the IBDs. Right? Show me someone who has an IBD who is not sensitive to gluten and I will show you a very rare case, (just look at the statistics in our own test files). The exceptions are usually drug-induced cases, and in those cases, stopping the use of the drug typically stops the inflammation, if it's done before other sensitivities are allowed time to develop.
And examples based on "IBS" won't work, because if long-term enteritis (intestinal inflammation) is involved, then clearly (by definition), you are talking about an inflammatory bowel disease instead. Remember, only serious, long-term intestinal inflammation can cause progressive loss of multiple enzymes.
So it might be possible, but it appears to be very, very unlikely, statistically. At least that's the way I see it.
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
So you are saying that the millions of people who suffer from "IBS" like symptoms ALL have an IBD? My cousin is a perfect example of someone who was tested for IBDs, and took the Enterolab test (and had no over antibody production). She KNOWS she gets gas, bloating , and pain when eating certain foods and that when she doesn't eat those foods, she feels fine. ????????
leah
leah
Inflammation is the determining criterion for an IBD, clinical symptoms are secondary to that. Remember that just because a GI doc fails to diagnose an IBD is not proof that an IBD is not present. Until relatively recently, unless the patient was an older woman, for example, a diagnosis of MC was extremely unlikely, simply because the doctors failed to look for the disease.
You're assuming that GI specialists actually know what they're doing, but the fact is that medicine is a very imperfect science. Why do you think that the average time from the onset of symptoms to an official diagnosis of celiac disease is still 9.7 years in this country? Celiac disease has been around for thousands of years. They should be able to diagnose it by now, but they still don't know how to diagnose the disease reliably until the small intestine looks like a battleground. Overall, they only diagnose a relatively small percentage of the total cases. That's ridiculous.
It's possible for the immune system to produce inflammation without antibodies. MCAD is a good example of this.
But my first question is, "Has she ever been tested for the ability to produce adequate amounts of gastric acid?"
Tex
You're assuming that GI specialists actually know what they're doing, but the fact is that medicine is a very imperfect science. Why do you think that the average time from the onset of symptoms to an official diagnosis of celiac disease is still 9.7 years in this country? Celiac disease has been around for thousands of years. They should be able to diagnose it by now, but they still don't know how to diagnose the disease reliably until the small intestine looks like a battleground. Overall, they only diagnose a relatively small percentage of the total cases. That's ridiculous.
It's possible for the immune system to produce inflammation without antibodies. MCAD is a good example of this.
But my first question is, "Has she ever been tested for the ability to produce adequate amounts of gastric acid?"
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
I don't think so, but she has no problem digesting the foods that are not on the FODMAP list. She feels great when she adheres to no FODMAP foods. This week is the first "test" of something she would like to eat on the list... garlic. The next thing is avocado.
We went camping together this weekend and between her list and my list, it was almost comical. We both did well and i felt great. No bathroom issues :)
leah
We went camping together this weekend and between her list and my list, it was almost comical. We both did well and i felt great. No bathroom issues :)
leah