VLS#3 (from the food Doc)
Moderators: Rosie, Jean, CAMary, moremuscle, JFR, Dee, xet, Peggy, Matthew, Gabes-Apg, grannyh, Gloria, Mars, starfire, Polly, Joefnh
VLS#3 (from the food Doc)
When the eagles are silent, the parrots begin to jabber"
-- Winston Churchill
-- Winston Churchill
Thanks for the heads up! So great to have so many eyes looking for new stuff.
Great article
Particulalrly-
" This means it is possible that some who have undergone testing through Enterolab may in fact be carrying at least a minor subunit of these high risk genes DQ2 and/or DQ8 that does predispose to celiac disease rather than just non-celiac gluten sensitivity."
Wowy - wow -wow. For a leading researcher to put in print what so many of us have found on our own is real progress.
My best
Matthew
Great article
Particulalrly-
" This means it is possible that some who have undergone testing through Enterolab may in fact be carrying at least a minor subunit of these high risk genes DQ2 and/or DQ8 that does predispose to celiac disease rather than just non-celiac gluten sensitivity."
Wowy - wow -wow. For a leading researcher to put in print what so many of us have found on our own is real progress.
My best
Matthew
Very interesting. Thank you for the link. I'll be interested in his future reports.
Correct me if I'm wrong, but haven't we assumed that we don't have an increased risk of cancer because we don't have celiac sprue? If the above quote is true, could we be at increased risk if we don't control this disease? We definitely need more research done. I bet there'd be a lot more of us if the people with IBS were accurately diagnosed.This means it is possible that some who have undergone testing through Enterolab may in fact be carrying at least a minor subunit of these high risk genes DQ2 and/or DQ8 that does predispose to celiac disease rather than just non-celiac gluten sensitivity.
You never know what you can do until you have to do it.
Gloria,
You've raised some very interesting questions. Of course, some of us actually do carry one or both of the celiac genes, and some of us even carry a double copy of one, (IOW, they inherited celiac genes from both parents). That doesn't guarantee that any of us will actually develop celiac sprue, however, (thought we are obviously susceptible).
We've always assumed that untreated MC might carry an increased risk of lymphoma, the way that celiac disease does, though we've been inclined to sort of downplay that risk, since it's slight. We've probably felt that way because there is very little documentation of such a risk, in the literature. (Of course, MC was first defined only a little over 20 years ago.) However, the thinking on the risks involved may be changing. A little research turned up this conclusion:
http://www.jcge.com/pt/re/jclngastro/ab ... 28!8091!-1
Prudence seems to suggest that yes, we definitely need to do our best to try to control MC.
I suspect that you're quite right about the IBS situation. I really believe that the distinguishing feature between IBS and MC in many cases hinges on whether or not the GI doc bothers to take biopsies during a colonoscopy exam. If he does - the diagnosis is likely to be MC, and if he doesn't - the diagnosis is IBS, (by default).
Tex
You've raised some very interesting questions. Of course, some of us actually do carry one or both of the celiac genes, and some of us even carry a double copy of one, (IOW, they inherited celiac genes from both parents). That doesn't guarantee that any of us will actually develop celiac sprue, however, (thought we are obviously susceptible).
We've always assumed that untreated MC might carry an increased risk of lymphoma, the way that celiac disease does, though we've been inclined to sort of downplay that risk, since it's slight. We've probably felt that way because there is very little documentation of such a risk, in the literature. (Of course, MC was first defined only a little over 20 years ago.) However, the thinking on the risks involved may be changing. A little research turned up this conclusion:
which is from this report in the Journal of Clinical Gastroenterology:Conclusions: This study indicates that collagenous colitis may be the presenting clinical and pathologic feature of celiac disease. Diagnosis of collagenous colitis should lead the clinician to consider exclusion of underlying occult celiac disease.
http://www.jcge.com/pt/re/jclngastro/ab ... 28!8091!-1
Prudence seems to suggest that yes, we definitely need to do our best to try to control MC.
I suspect that you're quite right about the IBS situation. I really believe that the distinguishing feature between IBS and MC in many cases hinges on whether or not the GI doc bothers to take biopsies during a colonoscopy exam. If he does - the diagnosis is likely to be MC, and if he doesn't - the diagnosis is IBS, (by default).
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.